The Japanese Cancer Association and Debiopharm Group Present Doctors Oshima and Tanaka with the 2012 'JCA-Mauvernay Award' for their Innovative and Outstanding Cancer Research
LAUSANNE, Switzerland, September 21, 2012 /PRNewswire/ --
Debiopharm Group™ (Debiopharm), a Swiss-based global biopharmaceutical group of companies with a focus on the development of prescription drugs that target unmet medical needs and companion diagnostics, today presented the 'JCA-Mauvernay Award' to Doctor Masanobu Oshima from Kanazawa University for his research on 'The Role of Inflammatory Responses in Gastric Cancer Development' and Doctor Shinji Tanaka from the Tokyo Medical and Dental University, for his research on the 'Surgical Oncology to Develop Novel Targeted Therapies for Advanced Digestive Cancer'.
The ceremony was held during the General Assembly of the 71st Annual Conference of the Japan Cancer Association (JCA) in Sapporo, Japan on the following theme: 'Towards a new era and liaison of cancer research and life science'. Professor Tetsuo Noda, President of the JCA, and Dr Rolland-Yves Mauvernay, Founder and President of Debiopharm Group™ presented both recipients with the 2012 Award for their outstanding and innovative research in oncology.
'We congratulate both scientists for their hard work and accomplishments', said Rolland-Yves Mauvernay. 'They have set the standard for innovation in the field of gastric and digestive cancer. We hope their work will lead to major and more targeted medical discoveries leading to effective treatments in the years to come'.
Dr Masanobu Oshima works in the Division of Genetics at the Cancer Research Institute of Kanazawa University.
His research focuses on the construction of a unique transgenic mouse model system to investigate the role of inflammatory responses in gastric tumorigenesis. In most cancer tissues, the prostaglandin E2 (PGE2) pathway is induced, however how PGE2 itself induces tumorigenesis in cooperation with an activated oncogenic pathway such as wingless-type (Wnt) signalling activation remains unknown.